i saw that you spoke about toe spacer. do you think it can give important progress? i mean if it’s worth buying it. thanks @Santiago Figarola

I'm currently wearing R2 Large (gave R1s and R2s to family and friends), so now I'll be able to compare 👀 Let's see if I'm able to wear R3 regularly.

Anyone of you get extra hypersalivation when wearing the mouthguard? Does it go away?

Palatal expansion involves separating the midpalatal suture and stimulating bone remodeling in the maxilla. This process relies heavily on active osteoblasts to form new, stable bone in the expanded gap, ensuring long-term stability and preventing relapse. The fat-soluble vitamins A, D, and K2 (especially MK-4) work synergistically to optimize this remodeling: Vitamin A(Retinol) promotes osteoblast differentiation and upregulates RUNX2, the master regulator of osteoblastogenesis. Vitamin D improves calcium absorption, supports osteoblast maturation, and balances bone turnover. Vitamin K2 (MK-4) activates osteocalcin and MGP, directing calcium precisely into the new bone matrix while enhancing osteoblast proliferation and mineralization. These vitamins also boost expression of osteogenic markers (ALP, osteocalcin, type I collagen) and support healthy remodeling without excessive osteoclast activity. High-quality animal proteins deliver essential amino acids (glycine, proline, lysine) for collagen synthesis—the organic scaffold of bone—and elevate IGF-1 to further activate osteoblasts. Key minerals (zinc, magnesium, phosphorus, copper) in highly bioavailable forms from meats, organs, and dairy co-activate RUNX2, inhibit osteoclasts, and promote maxillary bone density. An animal-based diet (meats, organs, eggs, grass-fed dairy and fruit/honey) uniquely provides these nutrients in synergistic, highly absorbable forms and evolutionary ratios—far superior to plant-based sources, where fat-soluble vitamins are less effective or scarce (MK-4 is almost exclusively animal-derived). Chewing denser animal foods (meats, tendons) adds mechanical stimulation to osteoblasts, enhancing remodeling signals and amplifying nutritional benefits for stable expansion. During active expansion and retention phases, an animal-based approach maximizes osteoblast activity, optimizes bone gene expression (e.g., RUNX2), ensures proper mineralization of new bone, and supports lasting skeletal changes in the palate.