@Steven Goodspeed yes I mean to send me a message on here directly

That looks right. I’ve just finished linking the Skool payment option, and I’ll be recording content and getting everything organized in the courses section. I’m still working through a few structural details around how the information is housed and how payments are processed, so there may be a short delay. Once everything is ready, I’ll pin an announcement. Thanks so much for the support.

I have a 10.5-year-old English Bulldog, and anyone who’s lived with a dog long enough knows how deeply you can care about their wellbeing. I have a real soft spot for dogs, especially when I see people doing everything they can to help them feel better. From what you’ve shared I don’t think this dog has a persistent allergy problem. What you’re seeing is post-corticosteroid physiology, which is a predictable biological state after long-term cortisone use. When this isn’t understood, people keep adding supplements and peptides with good intentions and end up slowing recovery. When the biology is understood, the path forward becomes much simpler. Months of cortisone disrupt three systems at the same time: mitochondrial electron flow, redox balance, and immune discrimination. Cortisone suppresses mitochondrial biogenesis and oxidative phosphorylation, so electrons don’t move cleanly through the system. They leak, oxidative stress rises, and antioxidant signaling is reduced. At the same time, immune cells lose their ability to clearly distinguish what should be tolerated from what should be attacked. When steroids are stopped, immune signaling often rebounds in a noisy, uncoordinated way. Mast cells and gut immune cells start reacting to normal inputs. This gets labeled as “ongoing allergy,” but it’s actually loss of immune discrimination driven by redox imbalance. The gut sits at the center of this, especially in dogs. Dogs have a shorter digestive tract, faster transit time, and higher sensitivity to microbial metabolites. Steroids thin the mucus layer, deplete phospholipids like phosphatidylcholine, impair epithelial mitochondrial density, and loosen tight junctions. That means the gut leaks signals, not just material, and those signals drive immune activation in the skin and elsewhere. This is why a dog can feel better internally before looking better externally. Nervous system and mitochondrial recovery happen first. Skin and coat are slow-turnover, redox-sensitive tissues and always lag behind.

@Daniel Enderli I give Jeter 500mcg to start and I have gone higher. For the lactulose I have my doctor call it in for me. Keep the dose low. Jeter gets 3ml and he is 68lbs. Keep us posted on how the pup is doing 🙏

@John O'Mahony This is one of those areas where context really matters. Balance Oil (with ALA and linoleic acid) isn’t something I’d call universally “good” or “bad” it’s situational. Whether it’s appropriate depends largely on your current omega-3 : omega-6 ratio, how those fats are being metabolized, and what your broader health picture looks like. Diet history, inflammatory load, mitochondrial function, insulin sensitivity, gut health, and even genetics all influence whether adding ALA and linoleic acid will be supportive or problematic. For some people it helps restore membrane balance and signaling; for others it can worsen inflammation or redox stress if the system is already skewed. That’s why I don’t use a one-size-fits-all answer here. The ratio and context matter far more than the product itself. Once those pieces are clear, it’s much easier to decide if Balance Oil makes sense or if something else is a better fit.

@Curtis Smith Thank you for sharing this. Have you run a Prodrome? I would be curious how the results compare.

@Tim Chaikovsky The difference between a ketone monoester and taking the same amounts of BHB and 1,3-butanediol separately comes down to physics, timing, and metabolic control, not just ingredients. In a monoester like KetoneAid, BHB is chemically bound to 1,3-butanediol through a covalent ester bond. That bond means the two molecules behave as one unified structure as they move through the body, rather than as two independent compounds. From a physics standpoint, that ester bond shares electron density and lowers polarity, which makes the molecule more stable and more membrane-permeable. Importantly, the bond does not break randomly. It requires enzymatic cleavage by esterases, primarily in the gut and liver. This creates a controlled, rate-limited release of energy rather than an immediate flood. In practical terms, the ester acts like a time-released metabolic packet that only opens when and where the body is equipped to handle it. Once cleaved, you get two effects in sequence. Free BHB enters circulation quickly and provides immediate ketone signaling and fuel. At the same time, the 1,3-butanediol is routed through the liver, where it is converted into additional BHB via alcohol dehydrogenase. This produces a second, delayed wave of ketones. The result is a smoother and more predictable rise in ketone levels, a more orderly shift in NAD⁺/NADH balance, and a cleaner increase in mitochondrial membrane potential without overshooting redox capacity. When BHB and 1,3-butanediol are taken separately without the bond, the system behaves very differently. Free BHB enters circulation immediately, often spiking quickly and relying heavily on monocarboxylate transporters. Free 1,3-butanediol is absorbed and metabolized on its own timeline. These two inputs are no longer coordinated. From a systems perspective, entropy is higher. Redox inputs arrive asynchronously, and the body has to reconcile two independent signals instead of one ordered sequence. This is why some people experience more variability, GI discomfort, lightheadedness, or inconsistent cognitive and energy effects when the compounds are unbound. Anecdotally, I’ve used both approaches with clients and I’ve seen many of the same real-world benefits from each. Both can raise ketones, improve perceived energy, and support cognition when used appropriately. For the majority of people, either approach can “work.” However, when you get truly granular, there is a meaningful difference in how they are metabolized. The esterified form tends to produce a more consistent response, smoother redox shifts, and fewer side effects, especially in sensitive or highly stressed systems.

KE4 is the true monoester. Kinetik Pro uses the same 1,3-butanediol-to-BHB ratio, but without the ester bond. Functionally similar, just… the less-pretty twin still effective, way more pleasant to be around, and dramatically better tasting.