Low energy is one of the most common complaints in medicine, coaching, and everyday life, yet it is one of the least precisely understood. People describe it as fatigue, burnout, brain fog, weakness, lack of motivation, or feeling “offline.” Athletes feel it when they cannot train. Patients feel it when they cannot work. High performers feel it when discipline no longer works. The problem is that “low energy” is not a diagnosis. It is a surface description of a system-level failure, and two people can experience nearly identical symptoms while the underlying biology is completely different. Treating them the same way helps one person and harms the other. To understand low energy correctly, you have to stop asking how to boost energy and start asking why energy is being limited in the first place. At the deepest level, there are two dominant failure modes. In one, the body cannot produce enough energy. In the other, the body is deliberately suppressing energy production. The first is mitochondrial damage, a capacity problem. The second is inflammatory inhibition, a regulatory decision. One is a broken engine. The other is a functioning engine with the brakes applied. Subjectively they feel similar. Biologically they are opposites. Everything that follows depends on recognizing which one you are dealing with. A simple model helps. Imagine the body as a car. The mitochondria are the engine. They take fuel and oxygen and convert them into usable energy in the form of ATP. Inflammation acts like the central control computer, deciding how much power the engine is allowed to produce. If the engine is damaged, pressing the accelerator does little. If the computer is limiting output, the engine could perform, but is being intentionally restrained. In both cases the car goes slow. Only one responds to pushing harder. Mitochondria exist inside nearly every cell and are responsible for producing ATP, the molecule that powers muscle contraction, nerve signaling, hormone synthesis, immune regulation, tissue repair, and cognition. Without adequate ATP, nothing in the body functions well. Energy production depends on intact mitochondrial membranes, functioning enzymes, proper redox balance, sufficient oxygen delivery, and a steady supply of micronutrients. When any part of this system is damaged, the maximum amount of energy the body can generate drops. This is not a motivational issue. It is a hard ceiling.

DHA is almost always introduced as a “fat.” An omega-3. Something you supplement for inflammation, brain health, or heart health. That framing is familiar, convenient, and incomplete. Calling DHA a fat is like calling copper “a metal used in pennies.” It isn’t wrong, but it misses the reason biology actually uses it. If you walk away from this article still thinking DHA is nutrition, you missed the point. DHA is not primarily fuel. It isn’t there to be burned for calories. It isn’t present in the brain because the brain “needs fat.” DHA is there because it has electrical properties that other lipids do not. And intelligence, perception, and performance are ultimately constrained by how electrons move. Once you see DHA through that lens, many things that seem disconnected suddenly line up. Why DHA concentrates in the retina. Why it dominates synaptic membranes. Why deficiency shows up as brain fog, visual fatigue, poor recovery, and nervous system instability long before structural disease appears. Why inflammation is often downstream, not causal. This article exists to correct a category error. DHA does not belong in the same conceptual bucket as dietary fats. It belongs in the category of materials biology uses to move information. Most lipids in the body are structurally useful but electrically quiet. Saturated fats are the clearest example. Their electrons are tightly localized. They form stable sigma bonds. They resist deformation. Electrically, they behave like insulation. That isn’t a flaw. Any system that uses electricity requires insulation. Structural lipids give membranes rigidity and durability. They keep compartments intact. But they do not move charge efficiently. Monounsaturated fats add some mechanical flexibility, but electrically they remain limited. One double bond introduces a small region of electron density, but electrons are still largely confined. These fats make membranes more fluid, but they do not fundamentally change how information moves along the membrane surface.

Just wanted to share this…. stumbled upon an extract called maizinol….. derived from corn. Has some pretty impressive effects for increasing deep sleep by up to an extra 30 minutes a night over the course of a few weeks which is pretty astounding. here’s the pubmed links if anyone is interested https://pmc.ncbi.nlm.nih.gov/articles/PMC12759108/ https://pmc.ncbi.nlm.nih.gov/articles/PMC9889011/ Interestingly it looks like cvs brand has the only product i could find that only has maizinol as the sole ingredient it’s called “deep sleep support” . Please chime in if anyone has tried this for sleep

The members have spoke and I listened....Most coaches talk about principles. Some share theory. Very few show you exactly what they do themselves. about to change that. I’m opening up my personal playbook, the protocol I run on myself, to show you how I structure my training, nutrition, supplementation, peptides, and recovery strategies to stay at the top of my game. This isn’t a “one-size-fits-all” plan. It’s the real system I use, built from: - Lab data and cellular feedback loops - Peptide science and mitochondrial optimization - Periodized training matched to performance goals - Nutrition timing dialed to physiology, not fads You’ll see the exact tools, dosages, timing, and reasoning I use and how I adjust based on metrics, recovery, and results. If you’ve ever wondered how a coach integrates the science into a living, breathing system… this is your chance to see it in action. Drop a 🔥 below if you want to see the full breakdown of The Coach’s Protocol.I will likely do this as a webinar. Let me know your thoughts who would be interested in seeing this to kick off our monthly case study feature.

This is my baseline, year round supplement stack focused on general health, longevity, and performance. I figure I’d share with the community. I also run a separate sleep stack, Gut health and a PED support stack when applicable, but everything below is what I consider foundational and evidence backed. 1.Creatine Monohydrate Benefits: • Strength and power output • Cognitive support • ATP buffering • Neuroprotection Dosage: 6 g daily 2. Fiber Powder (Psyllium Seed) Benefits: • Gut health and motility • LDL cholesterol reduction • Improved glucose control Dosage: 6 g daily 3. Omega-3 Fish Oil Benefits: • Anti-inflammatory support • Lipid profile optimization • Brain and cardiovascular health Dosage: • EPA: 2,760 mg • DHA: 1,240 mg 4. Vitamin D3 + K2 (MK-7) Benefits: • Bone health • Immune function • Proper calcium handling and cardiovascular support Dosage: Vitamin D3: 5,000 IU Vitamin K2 (MK-7): 180 mcg 5. BioActive B-Complex Benefits: • Energy metabolism • Methylation support • Homocysteine regulation • Cognitive and nervous system function Dosage: ½ serving (1 capsule) 6. Lutein + Zeaxanthin Benefits: • Eye and retinal health • Blue-light protection • Reduced oxidative stress • Long-term visual longevity Dosage: Lutein: 20 mg Zeaxanthin: 4 mg 7. Astaxanthin Benefits: • Mitochondrial and membrane level antioxidant • Supports skin, eyes, joints, heart, and brain Dosage: 12 mg daily 8. Magnesium Glycinate Benefits: • Improved sleep quality • Parasympathetic nervous system support • Muscle relaxation and recovery Dosage: 400 mg nightly Any questions drop them below and if anyone is interested in my sleep stack, PED stack or gut health stack lmk and I can make a post about them!