Hello warrior fam! 👋 Sorry I’ve been quiet for the last few months. The loss of fellow warriors in this group (and also in my community on Insta) has profoundly impacted me and lead to deep reflection about the unfairness and inequitable outcomes of cancer and mortality. I felt like any update I post would seem trivial and unhelpful during this difficult time for many of you. Although I REALLY didn’t want to, I ended up accepting some standard of care and integrating radiation into my latest intense protocol. The reason for this was financial stress, tumour growth for the first time in 2.5 years, and knowledge that my cancer cells have down-regulated Heat Shock Proteins (which in theory makes them more sensitive to radiation). I chose long-course, low dose radiation (25x1.8Gy) and integrated sensitisation strategies, such as daily HBOT, intermittent fasting and a few repurposed drugs prior. For recovery and minimising side effects I’ve been doing RLT, hydrogen therapy, CDS, while maintaining a high level of ketones as they have anti-inflammatory effects (especially β-OHB which reduces oxidative stress in healthy cells). Today was my final fraction. Initially I negotiated 15 due to fear of both the short and long term side effects, but as I’ve tolerated it so well I decided to have the full 25. I am very lucky to have been referred to a new radiation oncologist who has been open to personalising the treatment and genuinely curious about the potential of HBOT, fasting and the ketogenic diet. Radiation to the pelvis in women nearly always results in infertility and induces early menopause. General side effects also include proctitis, incontinence, rectal inflammation and pain, fibrosis, etc. I’m very shocked to report I am yet to experience any side effects, with the exception of the two days I decided not to fast, didn’t do HBOT, and drank coffee and ate a typical western breakfast. I had to know for sure that the adjunctive modalities I’ve implemented have been truly effective and not placebo, so this is why I experimented with what a “typical” patient (ignorant about the impact of GKI) would likely eat. On those two days (and two days after) I had extreme fatigue and a little irritation in the rectal mucosa. As a result of this, I did a 3 day consecutive fast to try to stop the progression of the side effects and lo and behold… they went away! Unfortunately, prolonged fasting wasn’t / isn’t possible as weight loss impacts the accuracy of the markings and measurements needed for precision radiation (and I certainly don’t want ionising gamma rays blasting non-tumorous rectal mucosa and causing a secondary cancer through oncogenesis of healthy epithelial cells).

Hey Warriors 🙂, I’ve been a bit quieter lately as we’ve been working behind the scenes on something important: My mum completed the Astron Health multi-omic analysis, and the results have finally arrived. Next week we meet Astron’s integrative oncologist to go through the full interpretation — but I want to share the key insights now, because this is something that can empower every person in this community. ⭐ What Astron Revealed About My Mum’s Cancer Her tumour is not random. It runs on specific biological pathways — almost like an engine with identifiable components. The major drivers they identified were: 🔥 FGFR2 → MAPK (Main Growth Engine) 🔥 VEGF / Angiogenesis (Blood Supply) 🔥 MMP15 (Invasion + Tissue Breakdown) 🔥 WNT Signalling (Stem-Cell Survival + Recurrence) 🔥 FANCA Mutation (DNA-Repair Weakness → ROS Sensitivity) And important update: ➡️ CTCs were only 2, which is very low and extremely reassuring. ⭐ This Report Was NOT Essential — But It Was Incredibly Helpful I want to make something clear to everyone: You do NOT need expensive molecular testing to build an effective, multi-pathway protocol. Over the past 1.5 years, I created my mum’s protocol by studying: - cancer signalling pathways - metabolic vulnerabilities - invasion markers - angiogenesis mechanisms - mitochondrial weaknesses - stem-cell biology And what shocked me was this: The Astron findings matched almost exactly what I had already built through research alone. This is HUGE for this community. It proves that: ✔ you can learn what drives your cancer ✔ you can build precision strategies without testing ✔ you can target the actual pathways behind the disease ✔ you do NOT need to shoot in the dark The report confirmed the work — it didn’t create it. ⭐ The One Pathway We Had Not Fully Targeted: RAS → MAPK Astron’s report highlighted one important component: Atorvastatin This inhibits RAS prenylation, which is the top upstream switch that activates the MAPK pathway — the main engine of her tumour.

Hi everyone, I noticed some people have commented that they’re struggling to get their GKI below 2 and/or maintain it. So I thought I’d share some tips. I am day 6 into a 14 day fast. This time I am experimenting with a tea fast of matcha, dandelion root and graviola leaves - they all have very promising data indicating evoking of apoptosis in colorectal cancer cells (and other cancer cells too). They may prevent cancer cells using autolytic cell cannibalism to their advantage (whereby they eat surrounding cells in fermentation-driven stress situations) and give this advantage to T-Cells and NK cells to eat the cancer cells instead. Also, they may reduce glutamine’s capacity to be transported and converted to glutamate. Anyway, I have been able to maintain a GKI of 0.3-0.7 when I am in my “pulse” phase with the following strategies: 1. Fasting for 48 hours - sometimes dry, sometimes water-only and sometimes fat-only - they all have produced similar results, but dry fasting is king! 2. Don’t measure ketones and blood glucose within 2 hours of exercising or waking up and be disheartened - your glucose will be naturally higher as it’s the first fuel source drawn on for anaerobic activity (but ketones will eventually be used over time) and due to the “dawn effect” 3. Gluconeogenesis from protein can increase your blood glucose levels, as well as muscle cannibalism during fasting, so drop your protein intake and/or lift weights to prevent this (disregard the short blood glucose increase - this will stop once you’re fully depleted of glycogen). But don’t overdo it - aim to maintain rather than grow because hypertrophy will stimulate mTOR signalling and throw off autophagy. 4. Don’t only rely on ketone supplements and huge amounts of fat - teach your body to use its own fat because it is not just ketones but the metabolic process of ketogenesis that is effective in metabolic therapy. Also, if you eat a lot of fat and take a lot of ketone supplements, this may drive down your blood glucose reading but your insulin could still be high (which we can’t measure regularly and hyperinsulinemia is a driver of tumour growth). 5. If safe to do so, consider fasting to evoke deeper ketosis. For example, in the first day of fasting my GKI was 4.1. On the second day it was 3.2. Then on the third day, it drove down to 0.7 and has been 0.5-0.7 on days 4, 5 and 6. If you are worried about undesired weight loss or feel unwell when fasting, fat fasting is more gentle and won’t block autophagy as fat has zero impact on insulin. This includes bullet proof coffee, e.g. a little butter and MCT oil but NOT cream. 6. Monitor your GKI more closely after you break the fast - you should not go above 2 unless you’re eating too much protein or (like me) have a mindless, carb-addict relapse! 7. Get over the uncomfortable feeling that comes with going from being jacked to skinny as fuck. I totally get it because I used to be so muscular and with each fast I end up looking like I’ve just come out of a labour camp by the end. But at the end of the day, the longer you can go (safely of course and under medical monitoring or at least with a supportive friend or family member) the better your GKI outcome will be.

Hey guys, just wondering if anyone has had problems with liver dysfunction during their treatment. Dad has been on fenbendazole (222mg, 6 days a week) and ivermectin (1mg/kg) for six weeks with no issues. Started pembrolizumab/carboplatin 10 days ago and today had a blood test showing liver dysfunction with ALT around 1000, up from 40 2 weeks prior. The doctors are thinking it's autoimmune from the pembro, and treating as such. Anyone had liver function go off to that degree with fenben? Thanks

This group exists so no one has to walk this path alone. Share as much or as little as you feel comfortable: - Your diagnosis or a loved one’s journey - What brought you here - Questions you’re exploring - Therapies or strategies you’re researching - Or even a photo — faces help turn stories into connection Every share matters. It helps others feel less isolated and strengthens the collective wisdom of the group. 📌 Post Categories — How to Share To keep the community organised and easy to navigate, please choose the category that best fits your posts: 📢 Announcements: Official updates from the Admin team. 👋 Introductions: Say hello and share as much (or as little) as you’re comfortable with about your journey. 💬 General Discussion: Open conversation on cancer, healing, mindset, and community-related topics. 💡Research & Education: Research, studies, resources, protocols, and learning materials. ❓Q&A: Questions and thoughtful answers from the community. 📝 Journeys & Results: Personal stories, protocols being explored, progress updates and testimonials. 🎭 Miscellaneous: Anything that doesn’t clearly fit another category. If you’re unsure, don’t worry — just choose your best guess. The goal is sharing, learning, and supporting each other, not perfection.