I wanted to share something important with you — not as medical advice, but as part of our learning journey together ❤️ This post is shared for transparency and education only, not to suggest or recommend anything medically. This protocol was designed privately for my mum by Astron Health and is shared here purely for learning and discussion within our community. ✅ Everyone’s biology, cancer type, treatments, and risks are different ✅ Always work alongside your medical team My mum’s current protocol was built/personalised using molecular testing from her blood, done through Astron Health, who work with Exacta360. This kind of testing doesn’t just look at cancer type. It looks at what pathways are active, what the cancer appears to rely on, and what biological signals are dominant. In simple terms — it doesn’t ask: “What cancer is this?” It asks: “What is this cancer doing?” That shift changes everything. ✅ What This Kind of Testing Looks At From a simple blood sample, the analysis can look at things like: • inflammation signaling • growth and survival pathways • angiogenesis (blood vessel formation) • immune suppression markers • estrogen and hormone metabolism • metabolic stress pathways • mitochondrial vulnerability It gives a much clearer picture of the terrain. ✅ Something That Really Stood Out to Me What surprised me most was this… Almost everything Astron included was already part of our existing protocol. The core metabolic approach we’ve been using aligned very closely with what the molecular testing came back with — thanks in huge part to this community, Dr. Seyfried, and Jane McLelland. They added a few additional layers — such as: • atorvastatin • indole-3-carbinol • luteolin • apigenin • ursolic acid • propranolol But the foundation was already there. That was incredibly validating — not just emotionally, but scientifically. It confirmed that targeting the right pathways truly matters. ✅ Compounds That Came Up in Her Results Some of the natural compounds included were:

Hey Warriors 🙂, I’ve been a bit quieter lately as we’ve been working behind the scenes on something important: My mum completed the Astron Health multi-omic analysis, and the results have finally arrived. Next week we meet Astron’s integrative oncologist to go through the full interpretation — but I want to share the key insights now, because this is something that can empower every person in this community. ⭐ What Astron Revealed About My Mum’s Cancer Her tumour is not random. It runs on specific biological pathways — almost like an engine with identifiable components. The major drivers they identified were: 🔥 FGFR2 → MAPK (Main Growth Engine) 🔥 VEGF / Angiogenesis (Blood Supply) 🔥 MMP15 (Invasion + Tissue Breakdown) 🔥 WNT Signalling (Stem-Cell Survival + Recurrence) 🔥 FANCA Mutation (DNA-Repair Weakness → ROS Sensitivity) And important update: ➡️ CTCs were only 2, which is very low and extremely reassuring. ⭐ This Report Was NOT Essential — But It Was Incredibly Helpful I want to make something clear to everyone: You do NOT need expensive molecular testing to build an effective, multi-pathway protocol. Over the past 1.5 years, I created my mum’s protocol by studying: - cancer signalling pathways - metabolic vulnerabilities - invasion markers - angiogenesis mechanisms - mitochondrial weaknesses - stem-cell biology And what shocked me was this: The Astron findings matched almost exactly what I had already built through research alone. This is HUGE for this community. It proves that: ✔ you can learn what drives your cancer ✔ you can build precision strategies without testing ✔ you can target the actual pathways behind the disease ✔ you do NOT need to shoot in the dark The report confirmed the work — it didn’t create it. ⭐ The One Pathway We Had Not Fully Targeted: RAS → MAPK Astron’s report highlighted one important component: Atorvastatin This inhibits RAS prenylation, which is the top upstream switch that activates the MAPK pathway — the main engine of her tumour.

Hi everyone, I noticed some people have commented that they’re struggling to get their GKI below 2 and/or maintain it. So I thought I’d share some tips. I am day 6 into a 14 day fast. This time I am experimenting with a tea fast of matcha, dandelion root and graviola leaves - they all have very promising data indicating evoking of apoptosis in colorectal cancer cells (and other cancer cells too). They may prevent cancer cells using autolytic cell cannibalism to their advantage (whereby they eat surrounding cells in fermentation-driven stress situations) and give this advantage to T-Cells and NK cells to eat the cancer cells instead. Also, they may reduce glutamine’s capacity to be transported and converted to glutamate. Anyway, I have been able to maintain a GKI of 0.3-0.7 when I am in my “pulse” phase with the following strategies: 1. Fasting for 48 hours - sometimes dry, sometimes water-only and sometimes fat-only - they all have produced similar results, but dry fasting is king! 2. Don’t measure ketones and blood glucose within 2 hours of exercising or waking up and be disheartened - your glucose will be naturally higher as it’s the first fuel source drawn on for anaerobic activity (but ketones will eventually be used over time) and due to the “dawn effect” 3. Gluconeogenesis from protein can increase your blood glucose levels, as well as muscle cannibalism during fasting, so drop your protein intake and/or lift weights to prevent this (disregard the short blood glucose increase - this will stop once you’re fully depleted of glycogen). But don’t overdo it - aim to maintain rather than grow because hypertrophy will stimulate mTOR signalling and throw off autophagy. 4. Don’t only rely on ketone supplements and huge amounts of fat - teach your body to use its own fat because it is not just ketones but the metabolic process of ketogenesis that is effective in metabolic therapy. Also, if you eat a lot of fat and take a lot of ketone supplements, this may drive down your blood glucose reading but your insulin could still be high (which we can’t measure regularly and hyperinsulinemia is a driver of tumour growth). 5. If safe to do so, consider fasting to evoke deeper ketosis. For example, in the first day of fasting my GKI was 4.1. On the second day it was 3.2. Then on the third day, it drove down to 0.7 and has been 0.5-0.7 on days 4, 5 and 6. If you are worried about undesired weight loss or feel unwell when fasting, fat fasting is more gentle and won’t block autophagy as fat has zero impact on insulin. This includes bullet proof coffee, e.g. a little butter and MCT oil but NOT cream. 6. Monitor your GKI more closely after you break the fast - you should not go above 2 unless you’re eating too much protein or (like me) have a mindless, carb-addict relapse! 7. Get over the uncomfortable feeling that comes with going from being jacked to skinny as fuck. I totally get it because I used to be so muscular and with each fast I end up looking like I’ve just come out of a labour camp by the end. But at the end of the day, the longer you can go (safely of course and under medical monitoring or at least with a supportive friend or family member) the better your GKI outcome will be.

Prostate Cancer Gleason 3+4=7 (Jan 2025) - any success with combination of Keto and IVERMECTIN/Fenbendazole?. I'm doing Keto for last six months + D3+K2, Fish Oil, B12, Berberine and recently added Magnesium and Curcumin. My prostate size decreased from 40 (Jan 2025) to 31 (Oct 2025), and my PSA stopped growing over this time, but per MRI the lesion is increased in size from 1.0 (Dec 2024) to 1.8 (Oct 2025). I dropped 35 lbs. Stopped using Berberine, but my cholesterol is more than 300 now...used to be ~ 100. Any success using IVERMECTIN/Fenbendazole for similar problem? Any advise please