This patient developed significant redness, irritation and ocular surface inflammation while using topical glaucoma medication. At first glance, this may seem like a common ocular surface issue. However, persistent redness and irritation should prompt us to consider allergy or intolerance to the glaucoma drops themselves. In this case, the eyes cleared significantly after all topical glaucoma drops were stopped. No topical anti-inflammatory treatment was required. During this period, the IOP was managed with oral Diamox (acetazolamide) while the ocular surface recovered. Once the suspected medication-related reaction was identified, treatment options could be reassessed. Topical drops can be reintroduced cautiously, one at a time, to identify the offending agent, or SLT can be considered to reduce the need for topical medication. The patient was managed by Dr Ashvira Moodley and has also undergone selective laser trabeculoplasty (SLT). Key takeaway: When a glaucoma patient develops persistent ocular surface inflammation, consider whether the treatment itself may be contributing to the problem. Stopping the offending drops, maintaining IOP control and then individualising further treatment can help protect both ocular surface health and long-term glaucoma control.

By Dr Kashmira Rawjee Modern cataract surgery is refractive surgery. A refractive surprise refers to an unexpected residual refractive error after cataract surgery, where the achieved postoperative refraction differs from the planned target. Although uncommon with modern biometry and IOL calculation formulas, refractive surprises can still occur and may significantly impact patient satisfaction. Fortunately, when surprises occur, we have excellent solutions available. The main types of refractive surprise after cataract surgery are classified according to the residual refractive error, it can be myopic, hyperopia or parent as residual astigmatism. A refractive surprise may result from: • Unusual ocular anatomy (short or long eyes) • Prior corneal refractive surgery • Ocular surface disease affecting keratometry • Errors in biometry or lens selection • Effective lens position variability • Less commonly, postoperative IOL displacement In this weeks case, this patient experienced an unexpected hyperopic refractive surprise following routine cataract surgery with a visual acuity of 0.7. Although the surgery itself was uncomplicated, the postoperative refraction left the patient dissatisfied with their visual outcome. Rather than performing an intraocular lens exchange—which can be technically more challenging and carries additional risks once capsular fibrosis has developed—I elected to implant a supplementary (add-on) intraocular lens in the ciliary sulcus. What is an add-on IOL? A supplementary IOL is specifically designed to sit safely in the ciliary sulcus, in front of the original capsular bag IOL. It works by adding the required refractive power to correct the residual error while leaving the primary lens undisturbed. Advantages include: Precise correction of residual refractive error Preservation of the original capsular bag IOL Lower risk than late IOL exchange Reversible if future intervention is required Excellent visual outcomes in appropriately selected patients

By Dr Ashvira Moodley A patient recently presented with significantly reduced vision following eye trauma. Examination revealed a subluxed intraocular lens (IOL) — meaning the artificial lens implanted during previous cataract surgery had become displaced from its normal position. At presentation, the patient’s visual acuity was 0.1. Following careful assessment, surgery was performed to remove the unstable lens and implant a posterior iris-fixated Artisan lens (secondary IOL). At the 6-week postoperative visit, the patient’s visual acuity had improved to 0.8, demonstrating a significant restoration of visual function and lens stability. The video below demonstrates the implantation of the secondary intraocular lens (posterior Artisan IOL). What is a subluxed IOL? A subluxed IOL occurs when the lens implant shifts out of position. This can happen due to: - Eye trauma - Weakening of the structures that support the lens - Previous eye surgery - Certain eye conditions affecting the lens support system Symptoms may include: - Blurred or fluctuating vision - Double vision - Glare and halos - Visual distortion Early assessment is important, as timely intervention can help restore vision and prevent further complications.

By Dr Kashmira Rawjee This weeks case is a 53 year old female presenting with sudden, painless loss of vision in her left eye. Her visual acuity was reduced to 0.3 in the left eye. On fundus examination, we were greeted by widespread retinal haemorrhages, dilated tortuous veins, cotton wool spots, and optic disc swelling—the classic "blood and thunder" appearance of a Central Retinal Vein Occlusion (CRVO). CRVO typically presents with acute, painless unilateral visual loss, ranging from mild blurring to profound vision impairment. Non-ischaemic CRVO accounts for 75% of cases and results in mild to moderate loss of acuity. Ischaemic CRVO results in profound retinal ischaemia and vision loss to 6/60 or worse. Some patients may also report metamorphopsia or visual field defects. Risk factors include hypertension, diabetes mellitus, hyperlipidaemia, glaucoma, smoking, cardiovascular disease, and hypercoagulable states. In younger patients, an underlying inflammatory or thrombotic disorder should be considered. The consequences can be sight-threatening. Macular oedema is the most common cause of reduced vision, while retinal and iris neovascularisation can lead to neovascular glaucoma and permanent visual loss if not detected and treated promptly. Our patient started a course of Avastin injections for the macular oedema and will continue with treatment until the macula oedema resolves and her vision improves. Further management focuses on identifying and controlling systemic risk factors, monitoring for ischaemia and neovascularisation. Pan-retinal photocoagulation may be required when neovascularisation develops. As optometrists, you are often the first clinicians to identify CRVO. Timely diagnosis, appropriate systemic evaluation, and urgent referral can make a significant difference in visual outcomes.

By Dr Sachin Bawa This week’s case highlights an important complication of keratoconus — corneal hydrops. A young lady (30 years old), known with keratoconus, presented with sudden reduced vision in the right eye. She was initially referred with a possible corneal ulcer. Her background included scleral contact lens wear, with decent visual acuity. Clinical findings showed an oedematous right cornea, with the anterior segment OCT showing oedema in the stroma. A diagnosis of hydrops was made. UVAR) 0.05 ph 0.2 AVAL)0.7 Pachymetry R)244um Her corneas were noted to be very thin, making her unsuitable for corneal cross-linking. She had been managing well with scleral lenses prior to this event. Treatment of the hydrops included the injecting of sulfur hexafluoride (SF6) gas into the anterior chamber. The gas acts as a tamponade, helping to seal the break in Descemet’s membrane. Picture 3 shows her 1 day post op. Historically, hydrops were managed with sodium chloride eyedrops and antibiotic eyedrops. These cases often progressed to corneal scarring, with many patients ultimately requiring a full-thickness corneal transplant. In this case, intervention with SF6 gas injection saved the patient from a corneal transplant. SF6 gas is known to expand and can remain in the eye for approximately 7–14 days, and can assist in expelling the fluid from the cornea and promote healing. A great reminder that timely intervention can completely change the trajectory of these patients Picture 1: Cornea pre-op Picture 2: Cornea post-op picture 3: Slit lamp photo of right eye 1 day post op showing gas bubble.