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⚡ The short answer: It depends on your current mitochondrial health. SS-31 can make MOTS-c work better for some people… but it’s not a mandatory prerequisite. 🧱 What each one is “doing” SS-31 (Elamipretide) = mitochondrial structural repair Think: stabilizes the inner membrane/cardiolipin, lowers oxidative stress (ROS), improves ETC efficiency. MOTS-c = metabolic signaling & optimization Think: AMPK activation, better insulin sensitivity/glucose handling, mitochondrial biogenesis, “exercise mimetic” vibes. Analogy: SS-31 fixes the foundation 🧱 → MOTS-c upgrades the system ⚙️ (optimization works best when the “hardware” isn’t compromised). ✅ When SS-31before MOTS-c makes more sense: If you’re seeing signs of mitochondrial dysfunction/higher oxidative stress like: 😴 chronic fatigue / low energy 🏋️‍♀️ poor recovery 📉 metabolic decline (often 40+) 🍩 insulin resistance/metabolic issues 🤒 coming off illness/infection/prolonged stress 😵‍💫 poor sleep, high stress, processed diet, sedentary → sudden training ramp-up Suggested sequencing: SS-31 for ~4–8 weeks, then add MOTS-c (or overlap for synergy). ✅ When MOTS-c alone is probably fine: If you’re: 🧑‍🔬 younger (often <35) ⚡ already have solid energy + recovery 🥗 clean nutrition + good stress management 🏃 consistent training w/ good performance 🚫 no obvious metabolic dysfunction 🤔 If you’re unsure… A practical approach mentioned: try MOTS-c first → if results are “meh,” SS-31 may be the missing repair piece. 💬 Question for the group: If you’ve run either one (or both) — what did you notice most? Drop: age range + main goal + recovery/energy status 👇 📌 Educational discussion only — not medical advice.

I just added a brand new lesson to the classroom covering how people actually buy peptides from the grey market — and this one is especially important for those of you who are new here and have never purchased grey before. This lesson breaks everything down in plain English: 🧬 What the grey market really is 💸 Why U.S. peptide prices are so inflated 📲 How vendors operate (Telegram, WhatsApp, Discord) 🔍 How people vet vendors & verify legitimacy 🧪 How testing and COAs actually work 💳 Payments, shipping, customs & common issues If you’ve only ever bought peptides retail — or you’ve been curious but hesitant — this lesson will give you the full picture and help you understand how this world works before you ever decide to participate. 📚 Where to find it: Classroom tab → Vetted Vendor lesson 🎧 Prefer to listen instead? I also released this as a podcast + YouTube episode for easy consumption: 🎙️ Podcast: https://open.spotify.com/episode/7GEAnwFJTTzoEP4MaN2Wa0?si=vRpAPg7FRpaO-LMBic1D1w 📺 YouTube: https://youtu.be/rTCjldHie4g Whether you’re brand new to peptides or already deep in the optimization world, this lesson fills in a LOT of gaps that most people never explain. Highly recommend completing the written lesson and listening to the episode if you’re new here 🧠🌓

Alright, let’s talk plateaus — because I keep getting this question nonstop. If you’ve been on sema or tirz and the scale just… stopped moving… you’re not alone. And you’re not “doing it wrong.” This is biology. Why we plateau… When you lose weight, your body notices and fights back. Metabolism slows, hunger signals shift, and your body gets more efficient with calories. That’s metabolic adaptation — and it’s why a lot of people eventually stall (often around the 14–15 month mark on sema). What makes retatrutide different? Sema = GLP-1 Tirz = GLP-1 + GIP Retatrutide = GLP-1 + GIP + Glucagon That third receptor (glucagon) is the potential game-changer. In phase 2 data, people on the 12mg dose lost ~24.2% of body weight over 48 weeks, and what’s crazy is the weight-loss curve kept dropping — researchers noted a plateau hadn’t been reached yet. What glucagon is doing (in plain English)? Glucagon activity can help: - Increase energy expenditure (more burn at rest, not just less appetite) - Boost fat oxidation (more “use stored fat for fuel” signaling) - Increase thermogenesis (more heat production = more calories burned) - Reduce liver fat significantly (reported as high as ~82% at higher doses) Why this may help prevent plateaus? If you only reduce appetite, your body can adapt by slowing the engine down to match. With glucagon in the mix, you’re lowering intake and pushing burn up — which is basically fighting metabolic adaptation in real time. GLP-1 = appetite control GIP = nutrient handling / metabolic support Glucagon = keeps the metabolic “engine” running hotter Lifestyle still matters (non-negotiable) No matter what you’re using — sema, tirz, or reta — your results will hinge on: - Lift weights (2–3x/week minimum to protect muscle) - Protein first (low protein = muscle loss = slower metabolism) - Sleep (7–9 hours; poor sleep can stall you fast) - Stress management (chronic cortisol = stubborn fat storage)

We’ve been dropping some serious peptide breakdowns lately — full lessons, cheat sheets, and podcast episodes. Now I want your input 👇 Which compound should I deep dive into next? Whichever gets the most votes will be the next full deep dive: 📚 written lesson 🧾 one-page cheat sheet 🎙️ podcast episode