Hey Everyone The new platform is up and running. My DIY Bodybuilding program offers the following: 2 x courses on PEDs (1 for Males and 1 for Females)... this will be a growing library of videos that I will build upon so eventually you pretty much know everything I know lol. Access to ALL of my training plans...Plus the option to follow my training split. Feedback on your training videos MONTHLY live calls with yours truly And a Q&A thread if you ever need to ask any questions. Oh Oh, and you get discounted access to the 6 week fat loss challenge which will be help 2 x a week. Here's the link to join: https://whop.com/team-bates/diy-bodybuilding-lab/

Unlocked 😎🥳

Hey everyone I’ve been working in the background to update this platform. In the coming weeks I’ll be switching over to Whop. Upon doing so you will have instance access to the female and male PED courses as well as access to 6+ training programs where you can receive training feedback from me.

Ever wondered why fat storage shifts as women get older...especially around the midsection? The short answer: hormones. More specifically, estrogen. Estrogen’s Role in Fat Storage and Appetite Estrogen (estradiol in particular) plays a big part in regulating appetite, body weight, and fat storage. When estradiol levels are high, you’re more likely to feel satisfied after eating, which makes it easier to keep food intake under control. But as estradiol declines—especially during menopause—that sensitivity drops. Making it easier to overeat and gain weight. On top of that, estrogen influences where fat is stored. With normal levels, fat is more likely to settle in subcutaneous areas like your hips and thighs. This isn’t ideal for aesthetics if you hate stubborn lower body fat, but metabolically it’s less risky. When estradiol falls, fat tends to shift toward the abdomen as visceral fat—the deeper fat that surrounds organs and raises the risk of health issues. Metabolism Slows Down Too Declining estrogen doesn’t just affect appetite and fat distribution—it also lowers your metabolic rate. That means fewer calories burned at rest and a higher likelihood of weight creeping on. This is why many women notice fat loss feels harder in their 40s, 50s, and beyond compared to their 20s or 30s. The Good News Harder doesn’t mean impossible. You can still drop body fat—it just requires a smarter, more intentional approach. The basics still apply: - Lift heavy weights to hold onto muscle and keep metabolism higher. - Eat enough protein to support recovery and control hunger. - Do cardio to help with calorie burn and cardiovascular health. These aren’t just “nice-to-dos.” They’re the most effective tools to manage body composition when hormones aren’t working in your favor. If you want to dive deeper, here’s a solid breakdown of the science: Frontiers in Endocrinology, 2022

Drug dosing is often simplified into three main methods: fixed dosing, weight-based dosing, and body surface area dosing. Today, I want to focus on weight-based dosing, where the dose is calculated as milligrams per kilogram of body weight. At first glance, it makes sense—larger individuals would need a higher dose than smaller individuals because their bodies are bigger. However, this assumption has limitations. The assumption behind weight-based dosing is that all pharmacokinetic parameters, like how the body absorbs, distributes, metabolizes, and excretes a drug, scale proportionally with body weight. Essentially, doubling someone’s body weight would require doubling their dose. But the body doesn’t work that way. For example, enzymes which are responsible for metabolizing drugs, don’t increase in number or efficiency just because someone weighs more. Similarly, processes like filtration in the kidneys aren’t directly proportional to body size either. Take oxandrolone, for instance. A clinically studied dose for an 80 kg male is 20 mg, or 0.25 mg per kilogram. Using this formula, a 100 kg male would receive 25 mg, but this assumes that every extra kilogram of body weight increases the drug demand proportionally, which isn’t true. The liver enzymes processing the drug don’t multiply with weight, and neither do the androgen receptors the drug interacts with. So, what’s the takeaway? Weight-based dosing is a convenient tool, but it oversimplifies the complexities of human physiology. Drug metabolism depends on far more than body weight, and assuming a linear relationship can lead to inappropriate dosing.